introduction
Asciles is the symptom of disturbed water and electrolyte metabolism in cirrhosis of ihc liver. A variety of pathogenetic factors have been considered as possible causes.1 Whilst ascites was originally thought to result mainly from portal hypertension and
II fall in oncotic pressure, due to a disturoance of albumin synthesis, it is now
considered that insuBSiency of lymphatic drainage and, to a lesser exrenf nyper-
aldosteronism are the reasons for the sodium retention and increased elimination of
potassium. This form of hyperaldosteronism is not so much the consequence of an
activation, of the angiotensin-aldosterone system, due to a reduction in the effective
blood volume, but rather the result of a decrease in the metabolic clearance rate of
aldosterone in the liver.2-7'9-1c| These findings led to the therapeutic use of

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